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Additionally, while some studies suggest that under inflammatory conditions, α7nAchR can express in microglia and exert anti‐inflammatory effects [17, 42], our findings contrastingly indicate that during the stroke recovery period, α7nAchR primarily manifests within neurons rather than microglial cells across both the cortex and hippocampus

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Neurotrophic signalling peptides (P21 / P021): studied for BDNF-mimetic activity at the TrkB receptor